Promising new animal research suggests a drug originally developed to treat diabetes significantly reverses memory loss and brain degeneration in mice with a rodent version of Alzheimer’s disease. If the same is proven true in humans, the drug could one day be used as a treatment for Alzheimer’s disease and other memory-related illnesses.
In the study, published online this week in Brain Research, scientists from Lancaster University in England used lab mice to test how effective a diabetes drug known as a triple receptor was in treating Alzheimer’s disease. The mice in the study were specifically created to express certain genes associated with Alzheimer's disease in humans. The researchers waited for the mice to age before giving them the drug, therefore giving their disease some time to develop and damage the animal’s brain. Once given the drug, the animals were then made to conduct a maze test designed to measure their memory.
Results revealed that after being given the drug, aged mice who already had signs of a rodent version of Alzheimer’s disease showed improved learning and memory skills. The results were also seen on a biological level, and these mice displayed reduced amounts of plaque buildup in the brain, a major hallmark of Alzheimer’s disease. In addition, these mice also had reduced levels of chronic inflammation in their brains, overall slower rates of brain nerve cell loss and increased brain nerve cell protection. Continue reading...
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In the study, published online this week in Brain Research, scientists from Lancaster University in England used lab mice to test how effective a diabetes drug known as a triple receptor was in treating Alzheimer’s disease. The mice in the study were specifically created to express certain genes associated with Alzheimer's disease in humans. The researchers waited for the mice to age before giving them the drug, therefore giving their disease some time to develop and damage the animal’s brain. Once given the drug, the animals were then made to conduct a maze test designed to measure their memory.
Results revealed that after being given the drug, aged mice who already had signs of a rodent version of Alzheimer’s disease showed improved learning and memory skills. The results were also seen on a biological level, and these mice displayed reduced amounts of plaque buildup in the brain, a major hallmark of Alzheimer’s disease. In addition, these mice also had reduced levels of chronic inflammation in their brains, overall slower rates of brain nerve cell loss and increased brain nerve cell protection. Continue reading...
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